نتایج جستجو برای: globin mutation

تعداد نتایج: 297002  

درخشنده, جلال, زینلی, سیروس, طاهری, سحر, مرتضوی, یوسف,

Background and Objective: B-thalassemia is an autosomal recessive disease characterized by reduction or complete absence of b-globin gene expression. It has been estimated that more than 2,000,000 carriers as well as 20,000 patients affected with b-thalassemia are living in Iran, a country with more than 70 million population and great ethnic diversity. In this study we aimed to find out the b-...

Journal: :Nucleic acids research 1990
M C Barton M F Hoekstra B M Emerson

We have generated a site-specific 17 bp insertion within a 38 kb chick globin gene cluster by employing the recombination abilities of Saccharomyces cerevisiae. This gene cluster contains four beta-type globin genes which share a high degree of sequence homology. In this procedure, a small fragment of beta A-globin DNA containing a 17 bp insertion is subcloned into a URA3-based yeast integratin...

2011
Mohammad Hamid Frouzandeh Mahjoubi Mohammad Taghi Akbari Hossein Khanahmad Fatemeh Jamshidi Sirous Zeinali Morteza Karimipoor

Background: In the previous study, we have shown that the presence of A allele at position -588 in γ-globin gene was highly frequent and closely associated with fetal hemoglobin elevation among β-thalassemia intermedia patients. Therefore, we decided to investigate whether this allele (A allele at -588) could result in an increase in γ-globin gene expression to ameliorate the severity of the di...

2017
Mohammad Hamid Frouzandeh Mahjoubi Mohammad Taghi Akbari Hossein Khanahmad Fatemeh Jamshidi Sirous Zeinali Morteza Karimipoor

Background: In the previous study, we have shown that the presence of A allele at position -588 in γ-globin gene was highly frequent and closely associated with fetal hemoglobin elevation among β-thalassemia intermedia patients. Therefore, we decided to investigate whether this allele (A allele at -588) could result in an increase in γ-globin gene expression to ameliorate the severity of the di...

2005
George F. Atweh Corinne Wong Robin Reed Stylianos E. Antonarakis Ding-er Zhu Prahbat K. Ghosh Tom Maniatis Bernard G. Forget Haig H. Kazazian

A G to T transversion at the fifth nucleotide of the first intervening sequence (IVS-1 ) of the $-globin gene has been identified in cloned fi-thalassemia genes of two unrelated individuals. one of Mediterranean and the other of Anglo Saxon ancestry. In each patient the mutation was present in a different globin gene framework. defined by intragenic restriction site polymorphisms. thereby sugge...

Journal: :Blood 1980
A Deisseroth U Bode R Lebo A Dozy Y W Kan

We have succeeded in isolating hybrid mouse erythroleukemia cell clones from a patient with hemoglobin H disease, which exhibit either deletion or nondeletion mutations of the human alpha-globin genes. Analysis of one of these hybrid clones that had retained a human chromosome 16 from the patient's cells showed that both human alpha-globin had been deleted. Several clones of another hybrid cell...

Journal: :international journal of hematology-oncology and stem cell research 0
ebrahim miri-moghaddam sara bahrami majid naderi ali bazi morteza karimipoor

background: xmn-1 polymorphism of y g globin gene ( hbg2 ) is a prominent quantitative trait loci (qtl) in β-thalassemia intermediate (β-ti). in current study, we evaluated frequency of xmn-1 polymorphism and its association with β-globin gene ( hbb ) alleles and hb f level in β-ti patients in sistan and balouchestan province, south-east of iran. methods: 45 β-ti patients were enrolled. hbb gen...

Journal: :Blood 2015
Megan D Hoban Gregory J Cost Matthew C Mendel Zulema Romero Michael L Kaufman Alok V Joglekar Michelle Ho Dianne Lumaquin David Gray Georgia R Lill Aaron R Cooper Fabrizia Urbinati Shantha Senadheera Allen Zhu Pei-Qi Liu David E Paschon Lei Zhang Edward J Rebar Andrew Wilber Xiaoyan Wang Philip D Gregory Michael C Holmes Andreas Reik Roger P Hollis Donald B Kohn

Sickle cell disease (SCD) is characterized by a single point mutation in the seventh codon of the β-globin gene. Site-specific correction of the sickle mutation in hematopoietic stem cells would allow for permanent production of normal red blood cells. Using zinc-finger nucleases (ZFNs) designed to flank the sickle mutation, we demonstrate efficient targeted cleavage at the β-globin locus with ...

Journal: :apadana journal of clinical research 2012
kaveh jaseb khodamorad zandian manizheh kadkhodaie hamid galehdari mohamad pedram

background & objectives: the researcher clarified that β/globin gene cluster haplotypes in patients with sickle cell anemia provide useful population data as predictors of the disease severity, gene flow, and the origins of sickle cell mutation in this region. materials and methods: a total of 150 subjects was investigated in two different groups for five polymorphism restriction sites of t...

Journal: :Molecular and cellular biology 1999
J C McDowell A Dean

We investigated the requirements for enhancer-promoter communication by using the human beta-globin locus control region (LCR) DNase I-hypersensitive site 2 (HS2) enhancer and the epsilon-globin gene in chromatinized minichromosomes in erythroid cells. Activation of globin genes during development is accompanied by localized alterations of chromatin structure, and CACCC binding factors and GATA...

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