نتایج جستجو برای: brca1 gene

تعداد نتایج: 1145741  

Journal: :Journal of the National Cancer Institute 2000
B A Werness P Parvatiyar S J Ramus A S Whittemore K Garlinghouse-Jones I Oakley-Girvan R A DiCioccio J Wiest Y Tsukada B A Ponder M S Piver

BACKGROUND The two-hit hypothesis for the genesis of cancer predicts that cancer can develop when the wild-type allele of a tumor suppressor gene is lost in an individual with a germline mutation in that gene. Neither loss of heterozygosity (LOH) for BRCA1 nor mutations of the TP53 (also known as p53) gene have been documented prior to invasion in ovarian cancers arising in women with germline ...

Journal: :Cancer research 2013
Jingxuan Shan Shoba P Dsouza Sasha Bakhru Eman K Al-Azwani Maria L Ascierto Konduru S Sastry Shahinaz Bedri Dhanya Kizhakayil Idil I Aigha Joel Malek Issam Al-Bozom Salah Gehani Stacia Furtado Edith Mathiowitz Ena Wang Francesco M Marincola Lotfi Chouchane

Although the linkage between germline mutations of BRCA1 and hereditary breast/ovarian cancers is well established, recent evidence suggests that altered expression of wild-type BRCA1 might contribute to the sporadic forms of breast cancer. The breast cancer gene trinucleotide-repeat-containing 9 (TNRC9; TOX3) has been associated with disease susceptibility but its function is undetermined. Her...

Journal: :Journal of insurance medicine 1996
M L Engman C A Pinkham

Anticipating that genetic tests designed to determine an individuals predisposition to disease would eventually become commercially available, and recognizing that the results of many of these tests may have mortality implications, in 1994 Lincoln National Reinsurance Companies began developing a methodology that would allow the mortality risk associated with a number of different genetic mutat...

2012
Francisco J. Ortega José M. Moreno-Navarrete Dolores Mayas Eva García-Santos María Gómez-Serrano José I. Rodriguez-Hermosa Bartomeu Ruiz Wifredo Ricart Francisco J. Tinahones Gema Frühbeck Belen Peral José M. Fernández-Real

CONTEXT Expression and activity of the main lipogenic enzymes is paradoxically decreased in obesity, but the mechanisms behind these findings are poorly known. Breast Cancer 1 (BrCa1) interacts with acetyl-CoA carboxylase (ACC) reducing the rate of fatty acid biosynthesis. In this study, we aimed to evaluate BrCa1 in human adipose tissue according to obesity and insulin resistance, and in vitro...

Journal: :Frontiers in bioscience : a journal and virtual library 2003
Meena Jhanwar-Uniyal

The BRCA1 gene was isolated in 1994; germline mutations of this gene are known to confer susceptibility to breast and ovarian cancer in high-risk families. Since its discovery, several mutations have been identified in this gene; these are scattered throughout the gene, and include insertion and deletion frameshifts, base substitutions, and inferred regulatory mutations. It role in the pathogen...

2013
Alexandra V. Stavropoulou Florentia Fostira Maroulio Pertesi Marianthi Tsitlaidou Gerassimos E. Voutsinas Olga Triantafyllidou Aristotelis Bamias Meletios A. Dimopoulos Eleni Timotheadou Dimitrios Pectasides Christos Christodoulou George Klouvas Christos Papadimitriou Thomas Makatsoris George Pentheroudakis Gerasimos Aravantinos Vassilis Karydakis Drakoulis Yannoukakos George Fountzilas Irene Konstantopoulou

Germline mutations in the BRCA1 and BRCA2 genes contribute to approximately 18% of hereditary ovarian cancers conferring an estimated lifetime risk from 15% to 50%. A variable incidence of mutations has been reported for these genes in ovarian cancer cases from different populations. In Greece, six mutations in BRCA1 account for 63% of all mutations detected in both BRCA1 and BRCA2 genes. This ...

Journal: :journal of genetic resources 2015
fereshteh mir mohammadrezaei

abstract accumulation of gene changes and chromosomal instability in response to cellular dna damage lead to cancer. dna damage induces cell cycle checkpoints pathways. checkpoints regulate dna replication and cell cycle progression, chromatin restructuring, and apoptosis. checkpoint kinase 2 (chk2) is activated in response to dna lesions. atm phosphorylate chk2. the activated chk2 kinase can p...

Journal: :Cancer discovery 2015
Sarah L Sawyer Lei Tian Marketta Kähkönen Jeremy Schwartzentruber Martin Kircher Jacek Majewski David A Dyment A Micheil Innes Kym M Boycott Lisa A Moreau Jukka S Moilanen Roger A Greenberg

UNLABELLED Deficiency in BRCA-dependent DNA interstrand crosslink (ICL) repair is intimately connected to breast cancer susceptibility and to the rare developmental syndrome Fanconi anemia. Bona fide Fanconi anemia proteins, BRCA2 (FANCD1), PALB2 (FANCN), and BRIP1 (FANCJ), interact with BRCA1 during ICL repair. However, the lack of detailed phenotypic and cellular characterization of a patient...

Journal: :Cancer prevention research 2013
Asher Y Salmon Mali Salmon-Divon Tamar Zahavi Yulia Barash Rachel S Levy-Drummer Jasmine Jacob-Hirsch Tamar Peretz

Approximately 5% of all breast cancers can be attributed to an inherited mutation in one of two cancer susceptibility genes, BRCA1 and BRCA2. We searched for genes that have the potential to distinguish healthy BRCA1 and BRCA2 mutation carriers from noncarriers based on differences in expression profiling. Using expression microarrays, we compared gene expression of irradiated lymphocytes from ...

Journal: :The Journal of biological chemistry 2010
Tapas Saha Jeong Keun Rih Rabindra Roy Rahul Ballal Eliot M Rosen

Inactivation of the breast cancer susceptibility gene BRCA1 plays a significant role in the development of a subset of breast cancers, although the major tumor suppressor function of this gene remains unclear. Previously, we showed that BRCA1 induces antioxidant-response gene expression and protects cells against oxidative stress. We now report that BRCA1 stimulates the base excision repair pat...

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