نتایج جستجو برای: brafv600e

تعداد نتایج: 835  

2018
Antoneicka L. Harris Samantha E. Lee Louis K. Dawson Laura A. Marlow Brandy H. Edenfield William F. Durham Thomas J. Flotte Michael Thompson Daniel L. Small Aidan J. Synnott Svetomir N. Markovic John A. Copland

Patient-derived tumor xenograft (PDTX) mouse models were used to discover new therapies for naïve and drug resistant BRAFV600E -mutant melanoma. Tumor histology, oncogenic protein expression, and antitumor activity were comparable between patient and PDTX-matched models thereby validating PDTXs as predictive preclinical models of therapeutic response in patients. PDTX models responsive and non-...

2013
Jahan S. Khalili Patrick Hwu Gregory Lizée

Immunosuppressive tumor microenvironments limit the efficacy of T cell-based immunotherapy. We have recently demonstrated that the inhibition of BRAFV600E with vemurafenib relieves interleukin-1 (IL-1)-induced T-cell suppression as mediated by melanoma tumor associated fibroblasts (TAFs). These results suggest that inhibitors of the MAPK pathway in combination with T cell-based immunotherapies ...

2016
Chenlei Shi Yong Guo Yichen Lv Abiyasi Nanding Tiefeng Shi Huadong Qin Jianjun He

OBJECTIVE To investigate the clinicopathological characteristics of papillary thyroid microcarcinoma (PTMC) for surgery by comparing the difference between PTMC and larger papillary thyroid carcinoma (LPTC). METHODS We analyzed the differences in the clinicopathological characteristics, prognosis, B-type RAF kinase (BRAF)V600E mutational status and expression of angiogenic factors, including ...

2011
Eftychia Oikonomou Michal Koc Vladimira Sourkova Ladislav Andera Alexander Pintzas

Documented sensitivity of melanoma cells to PLX4720, a selective BRAFV600E inhibitor, is based on the presence of mutant BRAF(V600E) alone, while wt-BRAF or mutated KRAS result in cell proliferation. In colon cancer appearance of oncogenic alterations is complex , since BRAF, like KRAS mutations, tend to co-exist with those in PIK3CA and mutated PI3K has been shown to interfere with the success...

2015
Dagmara Rusinek Michal Swierniak Ewa Chmielik Monika Kowal Malgorzata Kowalska Renata Cyplinska Agnieszka Czarniecka Wojciech Piglowski Joanna Korfanty Mykola Chekan Jolanta Krajewska Sylwia Szpak-Ulczok Michal Jarzab Wieslawa Widlak Barbara Jarzab Paula Soares

BACKGROUND The molecular mechanisms driving the papillary thyroid carcinoma (PTC) are still poorly understood. The most frequent genetic alteration in PTC is the BRAFV600E mutation--its impact may extend even beyond PTC genomic profile and influence the tumor characteristics and even clinical behavior. METHODS In order to identify BRAF-dependent signature of early carcinogenesis in PTC, a tra...

2017
Chen Wang Xin Zhang Hui Li Xin Li Yansong Lin

OBJECTIVE Current diagnosis of radioactive iodine (RAI)-refractory (RAIR) differentiated thyroid cancer (DTC) is based on the imaging technique, which is of a high cost. Serum thyroglobulin (Tg) is a sensitive and easily obtained biomarker. Hence, we aimed to assess the predicting value of quantitative response of Tg in earlier identifying the RAIR-DTC with pulmonary metastasis. PATIENTS AND ...

Journal: :Cancer research 2005
Jeffrey A Knauf Xiaolan Ma Eric P Smith Lei Zhang Norisato Mitsutake Xiao-Hui Liao Samuel Refetoff Yuri E Nikiforov James A Fagin

The BRAFT1799A mutation is the most common genetic alteration in papillary thyroid carcinomas (PTC). It is also found in a subset of papillary microcarcinomas, consistent with a role in tumor initiation. PTCs with BRAFT1799A are often invasive and present at a more advanced stage. BRAFT1799A is found with high prevalence in tall-cell variant PTCs and in poorly differentiated and undifferentiate...

Journal: :Nature Reviews Clinical Oncology 2019

نمودار تعداد نتایج جستجو در هر سال

با کلیک روی نمودار نتایج را به سال انتشار فیلتر کنید