نتایج جستجو برای: arsenic trioxide ato

تعداد نتایج: 21287  

2017
Jing-Yi Zhang Bin Zhang Min Wang Wei Wang Ping Liao Gui-Bo Sun Xiao-Bo Sun

Arsenic trioxide (ATO) is a potent anticancer agent used to treat acute promyelocytic leukemia. However, its cardiotoxicity limits ATO's widespread clinical use. Previous studies demonstrated that ATO may aggravate Ca2+ overload and promote endoplasmic reticulum stress (ERS). Salvianolic acid B (Sal B) is cardioprotective against ATO and enhances ATO's anticancer activities. The present study a...

2011
Zhu Yuan Fang Wang Zhiwei Zhao Xinyu Zhao Ji Qiu Chunlai Nie Yuquan Wei

BACKGROUND Chemo-resistance to cisplatin-centered cancer therapy is a major obstacle to the effective treatment of human ovarian cancer. Previous reports indicated that arsenic trioxide (ATO) induces cell apoptosis in both drug-sensitive and -resistant ovarian cancer cells. PRINCIPAL FINDINGS In this study, we determined the molecular mechanism of ATO-induced apoptosis in ovarian cancer cells...

Journal: :Iranian journal of allergy, asthma, and immunology 2012
Li Zhang Keng Li Li Bing Ma Su-bo Gong Gu-yi Wang Yi Liu Xiao-ying Ji Li Xu Shao-kun Liu Ping Chen Ruo-yun Ouyang Xu-dong Xiang

In traditional Chinese medicine, arsenous compounds, including arsenic trioxide (ATO), are often used to treat many diseases, which are safe and effective. Recently, studies have indicated that Th17- IL-17 involved in the pathogenesis and development of asthma. The goal of this study was to investigate the effect and mechanism of ATO on asthma, especially the Th17- IL-17 axis.We used oval bumin...

2015
Zhen Yue Lingzhi Zhong Yan Mou Xiaotong Wang Haiying Zhang Yang Wang Jianxin Xia Ronggui Li Zonggui Wang

In a previous study, we found that induced expression of Heme Oxygenase-1 (HO-1) is responsible for the resistance of human osteosarcoma MG63 cells to the chemotherapeutic agent arsenic trioxide (ATO). The present study was aimed at investigating the molecular mechanisms underlying the induction of HO-1 that occurs after exposure of MG63 cells to ATO. First, using RT-QPCT and Western-blot, we f...

2012
Kuan-Hung Lu Hui-Ju Lee Min-Li Huang Shang-Chih Lai Yu-Ling Ho Yuan-Shiun Chang Chin-Wen Chi

The objective of this study was to examine the potential of enhancing the antileukemic activity of arsenic trioxide (ATO) by combining it with a folk remedy, crude methanolic extract of Mucuna macrocarpa (CMEMM). Human leukemia cells HL-60, Jurkat, and Molt-3 were treated with various doses of ATO, CMEMM, and combinations thereof for 24 and 48 h. Results indicated that the combination of 2.5 μM...

2015
Ezhilarasi Chendamarai Saravanan Ganesan Ansu Abu Alex Vandana Kamath Sukesh C. Nair Arun Jose Nellickal Nancy Beryl Janet Vivi Srivastava Kavitha M. Lakshmi Auro Viswabandya Aby Abraham Mohammed Aiyaz Nandita Mullapudi Raja Mugasimangalam Rose Ann Padua Christine Chomienne Mammen Chandy Alok Srivastava Biju George Poonkuzhali Balasubramanian Vikram Mathews

There is limited data on the clinical, cellular and molecular changes in relapsed acute promyeloytic leukemia (RAPL) in comparison with newly diagnosed cases (NAPL). We undertook a prospective study to compare NAPL and RAPL patients treated with arsenic trioxide (ATO) based regimens. 98 NAPL and 28 RAPL were enrolled in this study. RAPL patients had a significantly lower WBC count and higher pl...

Journal: :Acta biochimica et biophysica Sinica 2011
Guangfen Xiao Xueyuan Tang Chenjiao Yao Chenghong Wang

The novel chrysin analog 8-bromo-7-methoxychrysin (BrMC) has been reported to induce apoptosis of various cancer cell lines. Arsenic trioxide (ATO) treatment induces clinical remission in acute promyelocytic leukemia patients. The combination of ATO with other agents has been shown to improve therapeutic effectiveness in vitro and in vivo. In this report, the mechanism of apoptosis induced by t...

2016
Xiaofan Li Xiaoyan Guan Fang Li Yuanzhong Chen Nainong Li

OBJECTIVE To investigate the cytotoxic effects of suberanilohydroxamic acid (vorinostat) in combination with arsenic trioxide (ATO) on the human NB4 cell line in vitro. METHODS The rates of cell proliferation following treatment with vorinostat with or without ATO were measured. Flow cytometry of Annexin-V/propidium iodide double-stained cells was used to measure apoptosis. Acridine Orange an...

Journal: :Cellular physiology and biochemistry : international journal of experimental cellular physiology, biochemistry, and pharmacology 2017
Jing-Yi Zhang Gui-Bo Sun Yun Luo Min Wang Wei Wang Yu-Yang Du Ying-Li Yu Xiao-Bo Sun

BACKGROUND/AIMS This study aimed to investigate whether Salvianolic acid A (Sal A) conferred cardiac protection against Arsenic trioxide (ATO)-induced cardiotoxicity in H9c2 cells by inhibiting MAPK pathways activation. METHODS H9c2 cardiac cells were exposed to 10 µM ATO for 24 h to induce cytotoxicity. The cells were pretreated with Sal A for 4 h before exposure to ATO. Cell viability was d...

2011
Shaquana Jones Clement G. Yedjou Paul B. Tchounwou

The treatment of acute promyelocytic leukemia (APL) has been based on the administration of all-trans retinoic acid plus anthracycline chemotherapy, which is very effective as first line therapy; however 25 to 30% of patients will relapse with their disease becoming refractory to conventional therapy. Therefore, the aim of the present study was to investigate whether arsenic trioxide (ATO) indu...

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