نتایج جستجو برای: alveolar epithelial

تعداد نتایج: 195783  

Journal: :American journal of physiology. Lung cellular and molecular physiology 2009
Richard L Auten S Nicholas Mason Kathryn M Auten Mulugu Brahmajothi

Hyperoxia disrupts postnatal lung development in part through inducing inflammation. To determine the contribution of leukocyte-derived reactive oxygen species, we exposed newborn wild-type and NADPH oxidase p47(phox) subunit null (p47(phox-/-)) mice to air or acute hyperoxia (95% O(2)) for up to 11 days. Hyperoxia-induced pulmonary neutrophil influx was similar in wild-type and p47(-/-) mice a...

2017
Christin Peteranderl Jacob I. Sznajder Susanne Herold Emilia Lecuona

The respiratory epithelium is lined by a tightly balanced fluid layer that allows normal O2 and CO2 exchange and maintains surface tension and host defense. To maintain alveolar fluid homeostasis, both the integrity of the alveolar-capillary barrier and the expression of epithelial ion channels and pumps are necessary to establish a vectorial ion gradient. However, during pulmonary infection, a...

2014
Hongbo Li Jinjin Zhang Xiaodong Song Tao Wang Zhi Li Dong Hao Xiaozhi Wang Qingyin Zheng Cuiping Mao Pan Xu Changjun Lv

BACKGROUND Acute interstitial pneumonia is a rare interstitial lung disease that rapidly progresses to respiratory failure or death. Several studies showed that myofibroblast plays an important role in the evolution of diffuse alveolar damage, which is the typical feature of acute interstitial pneumonia. However, no evidence exists whether alveolar epithelial cells are an additional source of m...

Journal: :American Journal of Physiology-Lung Cellular and Molecular Physiology 2016

Journal: :The Journal of clinical investigation 2016
Christin Peteranderl Luisa Morales-Nebreda Balachandar Selvakumar Emilia Lecuona István Vadász Rory E Morty Carole Schmoldt Julia Bespalowa Thorsten Wolff Stephan Pleschka Konstantin Mayer Stefan Gattenloehner Ludger Fink Juergen Lohmeyer Werner Seeger Jacob I Sznajder Gökhan M Mutlu G R Scott Budinger Susanne Herold

Influenza A viruses (IAV) can cause lung injury and acute respiratory distress syndrome (ARDS), which is characterized by accumulation of excessive fluid (edema) in the alveolar airspaces and leads to hypoxemia and death if not corrected. Clearance of excess edema fluid is driven mostly by the alveolar epithelial Na,K-ATPase and is crucial for survival of patients with ARDS. We therefore invest...

Journal: :Clinical and experimental immunology 2010
B Roth Z'graggen J Tornic B Müller-Edenborn L Reyes C Booy B Beck-Schimmer

Apoptotic cell death has been considered an underlying mechanism in acute lung injury. To evaluate the evidence of this process, apoptosis rate was determined in effector cells (alveolar macrophages, neutrophils) and target cells (tracheobronchial and alveolar epithelial cells) of the respiratory compartment upon exposure to hypoxia and endotoxin stimulation in vitro. Cells were exposed to 5% o...

2012
Martin Fronius Wolfgang G. Clauss Mike Althaus

The ability to breathe air represents a fundamental step in vertebrate evolution that was accompanied by several anatomical and physiological adaptations. The morphology of the air-blood barrier is highly conserved within air-breathing vertebrates. It is formed by three different plies, which are represented by the alveolar epithelium, the basal lamina, and the endothelial layer. Besides these ...

Introduction: Idiopathic pulmonary fibrosis (IPF) is a relentlessly progressive and usually fatal lung disease of unknown etiology for which no effective treatments currently exist. In the adults type I and II pneumocytes, forms Components of the alveolar epithelial cells. In this study, we investigated the effect of thalidomide on the alveolar epithelial cells (type I and II pneumocytes) in ...

Journal: :The Journal of infectious diseases 2002
Craig Murdoch Robert C Read Qibo Zhang Adam Finn

Recruitment of neutrophils into alveolar air spaces is an early event in the pathogenesis of pneumonia due to Streptococcus pneumoniae. This results from chemokines released by activated endothelial and epithelial cells and alveolar macrophages. Culture supernatants of 6 wild-type strains of S. pneumoniae, shown to contain choline-binding protein A (CbpA; clades A and B), induced release of che...

Journal: :Infection and immunity 2007
Joshua D Hall Robin R Craven James R Fuller Raymond J Pickles Thomas H Kawula

Francisella tularensis replicates in macrophages and dendritic cells, but interactions with other cell types have not been well described. F. tularensis LVS invaded and replicated within alveolar epithelial cell lines. Following intranasal inoculation of C57BL/6 mice, Francisella localized to the alveolus and replicated within alveolar type II epithelial cells.

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