نتایج جستجو برای: ژن tp63

تعداد نتایج: 16672  

Journal: :Cell 2014
Katherine A. Hoadley Christina Yau Denise M. Wolf Andrew D. Cherniack David Tamborero Sam Ng Max D.M. Leiserson Beifang Niu Michael D. McLellan Vladislav Uzunangelov Jiashan Zhang Cyriac Kandoth Rehan Akbani Hui Shen Larsson Omberg Andy Chu Adam A. Margolin Laura J. van’t Veer Nuria Lopez-Bigas Peter W. Laird Benjamin J. Raphael Li Ding A. Gordon Robertson Lauren A. Byers Gordon B. Mills John N. Weinstein Carter Van Waes Zhong Chen Eric A. Collisson Christopher C. Benz Charles M. Perou Joshua M. Stuart

Recent genomic analyses of pathologically defined tumor types identify "within-a-tissue" disease subtypes. However, the extent to which genomic signatures are shared across tissues is still unclear. We performed an integrative analysis using five genome-wide platforms and one proteomic platform on 3,527 specimens from 12 cancer types, revealing a unified classification into 11 major subtypes. F...

2012
Tobias Brandt Joel L. Kaar Alan R. Fersht Dmitry B. Veprintsev

Most proteins have not evolved for maximal thermal stability. Some are only marginally stable, as for example, the DNA-binding domains of p53 and its homologs, whose kinetic and thermodynamic stabilities are strongly correlated. Here, we applied high-throughput methods using a real-time PCR thermocycler to study the stability of several full-length orthologs and paralogs of the p53 family of tr...

Journal: :Journal of Investigative Dermatology 2022

Stem cell – like tumor propagating cells self-renew to drive clonal expansion or differentiate into post-mitotic without tumorigenic potential in squamous carcinomas. This fate choice is governed by a transcriptional network comprised of SOX2-PITX1-TP63 driven self-renewal and KLF4 dependent differentiation circuits. Yet, how stem switch from remains elusive. Here, we report that this bi-stable...

2012
Yuan Yao Marcia Bellon Shary N. Shelton Christophe Nicot

Background: Inactivation of p53 and reactivation of telomerase expression are two of the most common events in human cancer. Results: Here we report the mechanisms used by p53, p63, and p73 to suppress telomerase expression. Conclusion: p53, p63, and p73 suppress telomerase expression through distinct pathways that involve E2F, E-box, c-Myc, and NF-YB. Significance: This study provides new insi...

2013
B. Zwiebach

1 From inner products to bra-kets Dirac invented a useful alternative notation for inner products that leads to the concepts of bras and kets. The notation is sometimes more efficient than the conventional mathematical notation we have been using. It is also widely although not universally used. It all begins by writing the inner product differently. The rule is to turn inner products into bra-...

Journal: :Proceedings of the National Academy of Sciences of the United States of America 2006
William M Keyes Hannes Vogel Maranke I Koster Xuecui Guo Yi Qi Kristin M Petherbridge Dennis R Roop Allan Bradley Alea A Mills

Homology between p63 and p53 has suggested that these proteins might function similarly. However, the majority of data from human tumors have not supported a similar role for p63 in tumor suppression. To investigate this issue, we studied spontaneous tumorigenesis in p63+/- mice in both WT and p53-compromised backgrounds. We found that p63+/- mice were not tumor prone and mice heterozygous for ...

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