LYMPHOID NEOPLASIA AICAR induces apoptosis independently of AMPK and p53 through up-regulation of the BH3-only proteins BIM and NOXA in chronic lymphocytic leukemia cells

نویسندگان

  • Antonio F. Santidrián
  • Diana M. González-Gironès
  • Daniel Iglesias-Serret
  • Llorenç Coll-Mulet
  • Ana M. Cosialls
  • Mercè de Frias
  • Clara Campàs
  • Eva González-Barca
  • Esther Alonso
  • Verena Labi
  • Benoit Viollet
  • Adalberto Benito
  • Gabriel Pons
  • Andreas Villunger
  • Joan Gil
چکیده

1Departament de Ciències Fisiològiques II, Institut d’Investigació Biomèdica de Bellvitge (IDIBELL)–Universitat de Barcelona, L’Hospitalet de Llobregat, Spain; 2Departament d’Hematologia Clínica, IDIBELL-Institut Català d’Oncologia, L’Hospitalet de Llobregat, Spain; 3Servei d’Hematologia, IDIBELL–Hospital de Bellvitge, L’Hospitalet de Llobregat, Spain; 4Division of Developmental Immunology, Biocenter, Innsbruck Medical University, Innsbruck, Austria; 5Institut Cochin, Université Paris Descartes, Centre National de la Recherche Scientifique (CNRS), Unité Mixte de Recherche (UMR) 8104, Paris, France; 6Inserm Unité 567, Paris, France; and 7Servicio de Nefrología, Hospital Universitario Marqués de Valdecilla–IFIMAV, Santander, Spain

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AICAR induces apoptosis independently of AMPK and p53 through up-regulation of the BH3-only proteins BIM and NOXA in chronic lymphocytic leukemia cells.

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تاریخ انتشار 2010